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August 4th 06, 11:39 PM
>
> That's interesting. Hip dysplasia is a vitamin C deficit:


WRONG= any 10th grade biology student will tell you that primates
(humans) and guinea pigs are the only animals on the face of the earth
that need Vitamin C.

All other animals, including canis familaris (dogs), can make their
own Vitamin C..

"scurvy" is a disease that affects collagen in people who don't have
adeqaute Vit C.
It was discovered that British sailors who never had fresh fruit got
it. They gave them limes and it got better...hence british saliors=
limey's

TJ PALVINO

H[email protected]hotmail.com
August 5th 06, 05:14 AM
rec.pets.dogs.health,rec.pets.dogs.behavior,alt.an imals.dog,alt.med.veterinary,rec.pets.cats.health+ behav

HOWEDY tj,

> wrote in message
oups.com...
>
>>
>> That's interesting. Hip dysplasia is a vitamin C deficit:
>
> WRONG=

"WRONG", tj? That's ludicrHOWES!

You CAN'T SAY THAT on accHOWENT of I'm Jerry Howe, The
Sincerely Incredibly Freakin Insanely Simply Amazing
Grand Puppy, Child, Pussy, Birdy And Horsey Wizard and
being WRONG AIN'T WIZARDLY, tj <{) ; ~ ) >

Here's the SCIENTIFIC CITES, tj:

http://www.belfield.com/article5.html

Canine Hip Dysplasia (CHD), a crippling disease of the coxofemoral
joint (hip), was first observed and reported in 1945 by the late Dr.
Gary Schnelle. Dr. Schnelle was the staff radiologist at Angels
Memorial Hospital at Boston, Massachusetts and reported his
observations in The North American Veterinarian Journal and termed
the condition "Congenital Coxofemoral Subluxation".

Though "congenital", by definition, means "existing at
birth but not hereditary", Schnelle theorized the cause
to be a recessive gene. Today, this condition is associated
with the large breeds of canines, however Schnelle's initial
observations were in the smaller breeds i.e., cocker spaniels,
terriers, etc..

During the 1960's, Dr. Wayne Riser, a veterinary pathologist,
collaborated with a geneticist and postulated the concept that
CHD was "polygenetic with environmental overtones" and during
this time frame, the term "Canine Hip Dysplasia" was born.

There has been a string of professionals who have perpetuated the
heredity theory even until the present. We have been encouraged to
breed selectively, alter, and in some cases, euthanize the afflicted
canines.

This is what the veterinary experts have been insisting the dog
breeders and fanciers do to prevent CHD for the past fifty-two
years. Is it possible we have been on the wrong track for more
than a half century? One authority, of a veterinary teaching
institution in a personal correspondence to the author, admitted
the "experts" have failed to solve the CHD problem.

In 1976, The author published his observations ("Chronic Subclinical
Scurvy and Canine Hip Dysplasia") in his small animal practice through
the administration of vitamin C to prevent CHD. Whether this concept
has been accepted or not it has sparked controversy, and has some
veterinarians reassessing the archaic heredity theory. There are two
unanswered relevant questions the "experts" have failed to answer to
prove the heredity theory.

Why are we so loathe to believe the idea today that so many
of our killers today such as cancer and heart disease can
be prevented by getting enough of the substances found in
fresh fruits and vegetables and other healthy foods?

Perhaps in the past we had the excuse of a lack of evidence,
but today that dodge is no longer an acceptable excuse for
neglecting healthy practices. And Dr. Packer points out that
a healthy diet is not nearly enough, we need to supplement
our modern diets to get the optimum amounts of these antioxidants.

Dr. Packer explains that of the hundreds of known antioxidants
there are five which seem to be the most important, and which
operate as a network, meaning that they help balance and recycle
each other. Cells are essentially filled with water surrounded
by a fatty wall, so both fat and water soluble varieties are
important.

Two of the network antioxidants are water soluble (vitamin C
and glutathione), two are fat soluble (vitamin E and CoQ10),
and one (lipoic acid) is both fat and water soluble. The
importance of this latter team member has only been known in
the last decade. It may be the most important of all and most
daily supplement manufacturers don't even include it.

Here's another EXXXCELLENT study on dysplasia
and vitamin C: http://www.workingdogs.com/doc0039.htm

> any 10th grade biology student

The Sincerely Incredibly Freakin Insanely Simply Amazing
Grand Puppy, Child, Pussy, Birdy And Horsey Wizard never
took biology on accHOWENT of geometry was prerequisite
and HE wanted algebra II insetad, so HE compromised and
took geometry as nap time an told the school to keep their
algebra and science and went to work for Uncle Sam instead.

> will tell you that primates (humans) and guinea pigs are
> the only animals on the face of the earth that need Vitamin C.

Yeah. The same ignorameHOWESES likeWIZE tell us to give
EXXXCESSIVE vaccinations and toxic treatments. 90% of ALL
DIS-EASE are CAUSED by veterinary malpractice and stress
from mishandling and poor nutrition from feedin commercial
GARBAGE dog foods like Science Diet <{}; ~ ) >

> All other animals, including canis familaris (dogs),
> can make their own Vitamin C..

Here's the RESEARCH article from Purdue:
http://www.vet.purdue.edu/epi/dietrisk.htm

HIP DYSPLASIA simply is a lack of VITAMIN C in your dogs diet.
Almost all dogs in the U. S . are raised on commercial dog food-
with no NATURAL VITAMIN C.

True, the manufacturers add ascorbic acid, but this chemical
form of Vitamin C just isn't good enough. Your dog MUST get
Vitamin C in his diet on a daily basis from a natural source,
like fresh vegetables or RAW MILK or Vitamin C made from a
VEGETABLE SOURCE"

Recent research from Purdue SEZ that ascorbic acid added
to dog food INCREASES the chances of GVD (bloat, gastric
torsion) by 320%.

steve crane aka gaubster (his secret alias) is the NUTRITIONIST /
SALESMAN for Science Diet Dog Foods International <{) : ~ ) >

Lolajoker ASKED steve to comment on the recent report from Purdue.
HE WON'T ADDRESS the ISSUE on accHOWENT of: Science Diet PRESERVES
THEIR DOG FOOD with (DRUMROLL, PLEASE, Mr. Maestro):

***ABSORBIC ACID***

Ta Dah! "RIMSHOT: BANG~!" (Thank you, Mr. Maestro!).

Perhaps it's time for you to find HOWET where steve is these
days and join him on accHOWENT of HE AIN'T POSTIN HERE abHOWETS
noDogdameneDMOORE on accHOWENT of HE'S A FRAUD, like yourself.

The Sincerely Incredibly Freakin Insanely Simply Amazing
GRAND
Puppy, Child, Pussy, Birdy And Horsey Wizard <{) ; ~ ) >

-------------------------------------------------

So, you got any SCIENCE to back up what you think you know, tj?

The Sincerely Incredibly Freakin Insanely Simply Amazing Grand
Puppy, Child, Pussy, Birdy And Horsey Wizard AIN'T playin
grabass here abHOWETS, tj.

Careers an reputations will be DESTROYED based on the CASE
HISTORY DATA in The Sincerely Incredibly Freakin Insanely
Simply Amazing Grand Puppy, Child, Pussy, Birdy And Horsey
Wizards' Human And Animal Behavior Forensic Sciences Research
Laboratory Archives on Google and other fine UNMODERATED pubic
news group archive search engines <{}: ~ ( >

> "scurvy" is a disease that affects collagen in
> people who don't have adeqaute Vit C.

And it causes THE SAME PROBLEM as H.D. for the
same reason, on accHOWENT of they're THE SAME
DIS-EASE <{}; ~ ) >

The Sincerely Incredibly Freakin Insanely Simply Amazing
Grand Puppy, Child, Pussy, Birdy And Horsey Wizard AIN'T
a doctor or veterinarian or even a Ph.D. HE only IDENTIFIES
EXXXPOSES and DISCREDITS ignorameHOWES veterinary
malpracticioners and university behaviorists and of curse
ETHICKAL breeders and professional trainers who jerk choke
shock bribe crate intimidate surgically sexually mutilate
and murder innocent defenseless dumb critters <{): ~ ) >

> It was discovered that British sailors who never had fresh fruit got
> it. They gave them limes and it got better...hence british saliors=
> limey's

And THAT'S HOWE COME the COMERCIAL DOG FOODS add UNABSORBABLE
forms of vitamin C to their GARBAGE dog foods which coincidentally,
causes a 320% INCREASED RISK of GVD (BLOAT aka gastric torsion)
<{) : ~ ( >

> TJ PALVINO

Perhaps you'd like to talk abHOWET other 100%
CONSISTENTLY UNNECESSARY AVOIDABLE PREVENTABLE
EZily REVERSABLE IATROGENIC DIS-EASES the veterinary
malpracticioners don't want you to know abHOWET
so they can fund their "canine genome project",
like PRA, eh tj?:

PRA, like CHD (Canine Hip Dysplasia a vitamin C deficiency
like scurvy) is a metabolic / nutritional defect and SHOULD
NOT PRESENT as a clinical problem anyMOORE NHOWE that we
KNOW ALL abHOWET IT:

http://www.guidedogs.org.uk/fileadmin/gdba/images/downloads/CPRAisnta...

Central Progressive Retinal Atrophy - isn't anymore June 1999

The most exciting outcome of this work is that the supplementation of
vitamin E to the dog's diet can maintain the blood levels at
sufficiently high values to stop the disease in its tracks.

What has been established is that in dogs which developed this
disease, there is a defect present in the metabolism pathway of
vitamin E. In the clinically affected breeds we studied, the
serum levels of vitamin E are usually lower than normal and this
means that there is less vitamin E available to the retina.

The constituent chemical in vitamin E is an important antioxidant
substance and it helps protect retinal tissue from damage caused
by light. Thus, where there is insufficient vitamin E the retina
can be easily damaged. The greater the deficiency the quicker the
disease appears and the quicker it progresses.

The work we have done has shown that affected dogs are simply
not able to retain ingested vitamin E in their system and
after a meal there is a rapid decay in the blood levels.

In fact this disease is very similar to the disease which
affects the human retina and is called FIVE (Familial
Isolated Vitamin E deficiency). We believe the source of
the defect may be a deficiency of the transport factor
produced by the liver that is required to move vitamin
E around in the circulation.

Some twenty years ago I looked at what could only be
described as an epidemic outbreak of RPED in the Briard
breed and it was those findings which suggested that a
nutritional factor was probably involved in the aetiology
of the condition. At that time, some thirty percent of the
breed was affected with RPED to varying extent by six years
of age.

At the time I completed a limited assessment of possible
nutritional factors and was impressed with the similarity
of RPED and the retinal changes which occur in acute Vitamin
E deficiency. In a number of both normal and clinically
affected Briards there was an indication from the blood
work that vitamin E might be significant.

One of the disease scourges of the pedigree dog world is
blindness caused by a number of progressive degenerations
of the retina, grouped together under the term "Progressive
Retinal Atrophy'1 (PRA).

It is a term that strikes fear into the hearts of dog breeders
simply because of its severe effects and the fact that there is
no treatment available.

So what is PRA and why have I entitled this article in such a way?

Sadly, intensive breeding programmes designed to improve the
standard of pedigree dogs have, on occasion, managed to select
deleterious genetic material and that material is seen as disease.

Intensive breeding does not produce the genetic mutations themselves
but the selection involved increases the incidence of mutations in
the general population. Random breeding on the other hand dilutes
this incidence.

With this disease the age of onset is variable and, unlike PRA,
this is not related to the breed of dog involved. RPED has been
recorded as early as fifteen months of age, but it may not make
its appearance until the eighth year of life in the same breed.

It was this considerable variability in the age of onset and the
speed of progression within the same breed which I considered to
indicate that factors other than a genetic predisposition may be
at work.

There is no doubt that breed predisposition clearly demonstrates
a genetic input, but it would seem obvious that other non-genetic
factors could influence the age of onset and the speed at which
the disease changes occur.

-----------

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